I agree the key here is speed and amount of air. It's not either/or. Either it always kills OR it's not an air embolism. I think the medical experts have done a good job explaining how they came to their diagnosis. If a little less air entered circulation or a little more slowly, it will cause symptoms but not necessarily be fatal. (This is supported by literature.) If enough air has entered, it is fatal and the baby is unable to be resuscitated.
It is air in the arterial circulation that is usually fatal. As the air gets to the venous bed of the lungs, it gets filtered out. That's why in adults, small amounts of air in venous circulation are not fatal. Babies also have a communication between venous and arterial circulation (ductus arteriosis) that typically closes in the first few days of life. Being one day older may have made a difference for twin B, if the DA was closed or closing. There would be no way to verify that without having gotten an echocardiogram before the collapse (because collapse events can reopen the DA due to hypoxia) but there was absolutely no clinical reason to have gotten an ECG at that time.
Other thoughts, baby B... can someone point me to exactly what sort of trouble baby B was in at birth? Such as needed 30 second of chest compressions, or intubated for surfactant and a day on the ventilator? Both of those do not portend a baby who is going to be ill for a prolonged period of time or who is necessarily more frail or unstable.
Also, as far as not needing follow up care - after the air has been filtered out by the lungs (all blood eventually gets to the lungs), and if she was fortunate enough not to have had a stroke, it is plausible that she would have recovered quickly. In fact this may lend even more credence to the idea that it was an air embolism, because other causes for collapse (sepsis, etc) would have left her on a ventilator, possibly with vasoactive drips, etc, after the initial resuscitation.`