2:05pm
The trial is now resuming after the court adjourned for a lunch break.
Dr Dewi Evans will continue to give evidence.
2:07pm
Dr Evans continues to discuss the 'realistic risks' Child C could have faced.
One was feeding; Dr Evans says all premature babies require naso-gastric feeding.
If the babies cannot tolerate that, then it is clinical practice to administer nutrition via TPNs, via IV.
Child C was fed via the latter method, which was "the right thing to do".
2:09pm
Dr Evans said aspirates would be taken from the stomach prior to feeding.
He said dark bile aspirates could be a symptom of NEC or an obstruction, but it would need to be taken in context with other signs such as the baby's abdomen condition, and the general condition of the baby - and signs of a problem would be whether the heart rate would increase, the breathing rate would increase, and/or whether the oxygen would need to be increased.
2:15pm
Dr Evans said medical staff were aware to monitor Child C's abdomen and make regular notes.
He says there is one entry made in the nursing notes of 'black fluid' - not necessarily bile, but discoloured blood. That was to be 'monitored' and to 'keep an eye' on the baby's condition. It would not, in itself, be a concern.
For the 'one-off' vomit reading, Dr Evans says if there was something 'serious' going on, it would happen more often than once.
The four dark bile aspirate readings, each 0.5ml, are 'a tiny amount', Dr Evans tells the court.
"The good news is it's only 0.5ml. The other good news is the bile aspirate is not increasing [per reading].
"That is an indication the baby is not getting worse."
He said increasing readings would point to an obstruction, as would a distended abdomen.
Dr Evans says Child C's status was "under control".
2:18pm
Dr Evans says Child C was well for a '30-weeker' (in terms of gestational age).
A blood test for CRP had increased from 1 to 22-23 - 'not particularly high', the clinical was 'aware of this' and Child C was placed on antibiotics.
Child C's platelet count had fallen - which 'on their own don't tell you very much', but in combination with an x-ray was a 'non-specific marker pointing to an infection'.
2:21pm
Blood gas readings taken were 'within acceptable values', Dr Evans tells the court, and in terms of metabolic readings, Child C was a 'stable little baby'.
Dr Evans said all premature babies develop symptoms of jaundice.
"The good news with [Child C] is the jaundice levels were very satisfactory".
If Child C had signs of severe jaundice, he would have required phototherapy, Dr Evans explains.
2:25pm
Dr Evans says Child C had a lung infection, of pneumonia, which was "very common" in premature babies, and he was placed on antibiotics in advance of any test results.
Mr Johnson: "Did breathing issues have any direct cause for [Child C's] death?"
Dr Evans: "No."
Mr Johnson: "Did any feeding issues cause his collapse?"
Dr Evans: "No, that cannot explain his collapse either."
Mr Johnson: "Did the infection of pneumonia cause it?"
Dr Evans: "No - the infection was under control and being treated."
Dr Evans explains if the pneumonia treatment was not working, a number of markers would be shown. There would be an increase in heart rate (which did not occur, he says), an increase in respiratory rate - but that stayed the same.
Oxygen saturation levels stayed "absolutely where they should be", whereas in worsening pneumonia conditions those levels would fall.
2:28pm
Mr Johnson: "Did the jaundice/glucose issues cause his collapse?"
Dr Evans: "None at all."
Dr Evans says there were "no worrying trends" recorded in the notes.
"What was the cause of [Child C's] catostrophic collapse and death?"
Dr Evans says, initially, he did not have a conclusion to Child C's death.
He adds one complication is if the abdomen is filled with air.
Dr Evans: "If you get a significant injection of air into the stomach, it can cause splintering of the diaphragm."
As a result, a baby could collapse pretty quickly as they would suffocate.
Dr Evans says that was his conclusion for Child C.
2:31pm
Dr Evans says if the diaphragm is unable to move effectively, the lungs are unable to get fresh oxygen, and that causes the collapse.
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